What is the mechanism of action for Homatropine in ocular treatment?

Homatropine acts primarily as an anticholinergic agent, which means it inhibits the action of acetylcholine at muscarinic receptors in the eye. This results in two key effects: mydriasis (dilation of the pupil) and cycloplegia (paralysis of the ciliary muscle). Mydriasis allows for better visualization of the interior of the eye during examinations and diagnostic procedures, while cycloplegia helps to facilitate eye exams by temporarily paralyzing accommodation, allowing the clinician to obtain a more accurate assessment of refractive errors without the influence of the eye's ability to adjust focus.

Understanding the specific effects of homatropine on pupil dilation and accommodation is crucial for its use in ocular treatments, particularly in situations like cycloplegic refractions and the management of certain inflammatory conditions. This makes mydriasis and cycloplegia the most relevant mechanisms of action in its ocular use.